Vaccines & Cancer: Is There a Connection?

“On August 10th, 1998 our only child, Alexander, was diagnosed with the most common pediatric brain cancer, medulloblastoma. He was two years old. Our lives were shattered. The next six months became a race against time to try to understand the disease, find the appropriate treatment, and save Alexander.

After two brain operations Alexander recovered quickly. We wanted to give our son the most effective cancer therapy possible. After weeks of research, many conversations with parents who had children with brain cancer, and conversations with doctors from all over the world, we selected the Burzynski Clinic in Houston, Texas. We arrived there and incredibly we were turned away. Dr. Burzynski said he was not allowed to accept Alexander. I’ll never forget it. We sat in an examining room. Alexander was smiling at the doctor.

“Why can’t you take Alexander?” I asked Burzynski.

“The FDA dictates who I can and can’t accept,” Burzynski said.

Burzynski explained to us that the FDA would only allow him to accept children who had suffered through chemotherapy and/or radiation and still had “measurable tumor” left in their brains. Alexander hadn’t had either of these “world class treatments” but already endured two brain operations (16 hours of surgery in total) and was tumor free for the moment. He had paid a dear price to be tumor free. His optic nerves had been injured so that his big brown eyes were stuck pointing in opposite directions, he lost the ability to cry and laugh and he temporarily lost the ability to walk.

“Please accept my son. He’s only two years old. His whole life is in front of him. I know your treatment works. I’ve spoken to several parents whose children are here. They had malignant brain tumors like Alexander but now they’re alive and well. You have to treat my son,” I begged.

Dr. Burzynski said simply, “I am sorry but I can’t.” Burzynski was saddened but he was powerless. The FDA had made him turn away many children just like Alexander.

Chemotherapy was started soon after and Alexander died in my arms three months later.”

The above is part of written testimony to Congressman Dan Burton and the Government Reform Committee on Vaccines, held in 1999. The parents went on to outline a number of symptoms occurring after vaccines, that eventually led to a diagnosis of brain cancer. They believed his cancer was linked to the numerous rounds of vaccines he’d had as a baby [1].

They are not the only ones who suspect that vaccines played a part in causing cancer.

In 2001, a letter published in the Daily Mail, went as follows “My daughter had the MMR booster at four and her arm immediately swelled up and she started to feel unwell. Within six weeks, she was diagnosed as having leukaemia, and the doctors we spoke to accepted that the MMR jab was probably the trigger for the disease by overloading her immune system — though they believe she may have been already susceptible to the illness” [2].

It’s not just parent’s wondering. Some doctors and scientists, too, have obviously wondered.

In 1965, Dr. Michael Innis, an Australian pathologist and haematologist, wrote to The Lancet, and outlined how rates of leukemia in children at Brisbane Children’s Hospital between 1958 to 1964 showed a statistically significant association with diptheria-tetanus-pertussis vaccination [3].

In 1994, researchers found that MMR vaccination (among other things) increased the odds ratio of childhood acute lymphocytic leukemia [4].

Researchers in 2007 proposed a correlation between childhood leukemia and the introduction of widespread diptheria vaccination – “the significant peak-age (2–5 years) first appeared after 1940 in Great Britain. Since then, childhood leukemia has almost unchangeable incidence. In 1940 the introduction of immunization against diphtheria on a national scale was begun in Great Britain [5]”.

Nevertheless, the long-term studies required to prove whether vaccines increase cancer risk are not necessary for vaccine approval, nor does the CDC feel they are required…[6].

The following chart shows the incidence of childhood cancers in Australia [7].

Note that the most common age for childhood cancer is in the 0-4 years age group. Also note, that this is the same time period where the average child receives more than 40 different vaccines. The second most common age is in the 10-14 years age group, which coincides with the scheduled booster shots and HPV vaccines for secondary school. The least represented age group in cancer statistics, is the 5-9 years, which happens to coincide with a period where the average Australian child receives no vaccines, or, a yearly flu vaccine at the most [8].

It is also interesting to note that the most common type of cancer in children is acute lymphoblastic leukemia, or ALL [9]. This occurs when there is an overproduction of immature white blood cells in the bone marrow, which prevents the production of red blood cells [10]. It seems plausible that chronic activation of the immune system could potentially cause such a state of affairs – an hypothesis that has already been explored in the scientific literature [11-12]

I have already written here about the fact that excessive stimulation of humoral immunity (which includes antibody production – the aim of vaccination) results in suppression of cell-mediated immunity. This same immune system imbalance has already been shown to play a central role in facilitating tumour growth, invasion and metastasis [13].

In a study of oral cancer patients in Nigeria, those with cancer were found to have significantly higher levels of antibodies, than healthy controls [14]. Did the cancer cause the shift towards antibody production, or did the immune imbalance cause the cancer?

Actually, it was demonstrated as early as 1907, that an inappropriate immune response enhances tumour growth [15]. In the 1950’s, the phenomena of antibodies promoting tumour growth was labelled “immunological enhancement” [16].

Research published in the Journal of Infectious Diseases in 1988 found that one-year-old infants vaccinated with measles vaccine experienced a significant decrease in the level of alpha-interferon produced by lymphocytes. This marked reduction was still evident when the study ended a year later [17].

Interferons are a type of cytokine. These molecules communicate between cells to co-ordinate immune responses that help to expel pathogens. Interestingly enough, interferon therapy is now being used as a cancer treatment [18].

Now, obviously none of this proves that vaccines cause cancer, but until the CDC or others are convinced of the urgency of long-term studies in this area, we are left to surmise and hypothesize, and grieving parents are left to forever wonder. Given that the CDC has a large vested interest in vaccines, with dozens of vaccine-related patents [19]…it’s not likely to be anytime soon…


[1] Testimony of Raphaele Moreau-Horwin & Michael Horwin, Government Reform Committee – Vaccines; Finding the Balance Between Public Safety and Personal Choice. US House of Representatives, 12th August 1999.

[2] Letter, Daily Mail, 25th Jan, 2001.

[3] Innis MD, Letter to the Editor: Immunization and Childhood Leukaemia, The Lancet, 13th March 1965, i605.

[4] Buckley JD, Buckley CM, Ruccione K, et al, Epidemiological characteristics of childhood acute lymphocytic leukemia. Analysis by immunophenotype. The Children’s Cancer Group, Leukemia, 1994, 8(5):856-864.

[5] Ivanovski P, Ivanovski I, Childhood acute lymphoblastic leukemia is triggered by the introduction of immunization against diphtheria, Medical Hypothesis, 2007, 68(2): 324-327.

[6] CDC, Parents Guide to Childhood Immunizations, Part 4: Frequently Asked Questions, Accessed March 2019.

[7] Cancer Australia: Children’s Cancer Statistics, Accessed September, 2017.

[8] Ibid

[9] St. Jude Children’s Research Hospital, Acute Lymphoblastic Leukemia (ALL), Accessed March 2019.

[10] Poplack DG (1985) Acute lymphoblastic leukemia in childhood. In: Altman AJ (ed) The Paediatric Clinics of North America. Saunders Philadelphia, pp 669–697.

[11] O’Byrne KJ, Dalgleish AG. Chronic immune activation and inflammation as the cause of malignancy, Brit J Cancer, 2001, 85(4):473-83.

[12] Dalgleish AG, O’Byrne KJ. Chronic immune activation and inflammation in the pathogenesis of AIDS and cancer, Adv Cancer Research, 2002, 84:231-76.

[13] O’Byrne KJ, Dalgleish AG, Browning MJ, et al. The relationship between angiogenesis and the immune response in carcinogenesis and the progression of disease, Eur J Cancer, 2000, 36(2):151-69.

[14] Akinmoladun VI, Arinola OG, Elumelu-Kupoluyi T, Eriba LO. Evaluation of humoral immunity in oral cancer patients from a nigerian referral centre, J Maxillofac Oral Surg, 2013, 12(4):410-3.

[15] Flexner S, Jobling JW. Proceedings of the Society for Exp Bio Med. 1907. p. 461.

[16] Kaliss N. Immunological enhancement of tumor homografts in mice: a review. Cancer Res, 1958, 992-1003.

[17] Nakayama T, Maehara N, Sadaki K, Makino S. Long-term regulation of interferon production by lymphocytes from children inoculated with live measles virus vaccine, J Infect Dis, 1988, 158(6): 1386-1390.

[18] Cancer Research UK, Interferon (Intron A), Accessed March 2019.

[19] Google search of vaccine-related patents held by CDC,,ptss:g&num=100. Accessed March 2019.

7 Reasons Why Antibodies Can’t Possibly Provide Immunity

There is a massive vaccine industry that rakes in billions in profits, based on the belief that if you have antibodies, you are ‘protected’. Here’s 7 reasons why that belief needs a re-think…


There are numerous cases in the scientific literature, of people succumbing to illness, even though they had high antibody counts [1-3]. In fact, some of those had antibody titres 100x higher than what is considered sufficient to provide ‘immunity’. On the other hand, there are people with little to no antibody counts (and supposedly susceptible) passing through disease outbreaks completely untouched [4].

Actually, the discovery that antibodies are not responsible for immunity was made more than 80 years ago, by immunologist Dr. Merrill Chase, and his discovery was largely ignored by mainstream medicine, despite a long and illustrious career, and publishing more than 150 research papers [5].


According to vaccine logic, the more antibodies you have, the better, but in a NORMALLY functioning immune system, antibody production is tightly restricted (for good reason – more on that later). It’s now common knowledge that Vitamin D is necessary for a healthy immune system…but did you know Vitamin D LIMITS antibody production [6]? It begs the question why, if antibodies really are as vital as we have been led to believe…


The presence of prior antibodies has been found to ENHANCE some diseases. It’s called ‘antibody-dependant enhancement’ and, so far, it has been demonstrated to enhance dengue fever, zika virus, HIV, Ebola, and others [7-12].


Antibodies are created in the body as a last resort. It only occurs AFTER the cells have become infected. Remember the selling point of vaccines – about having a ‘primed’ immune system, so that antibodies could respond faster? Well, technically that’s true, but they neglected to mention that, even in a ‘primed’ immune system, antibodies are STILL not called into action, until after infection occurs [13]. Therefore, it’s a biological impossibility for antibodies to prevent infection, even in a ‘primed’ immune system.


By now, you may be wondering why the human body is designed to limit, restrict or delay antibody production. There’s a good reason for this – because antibodies are highly inflammatory and uncomfortable. Those unpleasant symptoms that you experience when ‘sick’ are not symptoms of disease, they are the result of antibodies. Antibodies place a large burden on the body’s excretory systems and, if not excreted in a timely manner, they conglomerate and form ‘antibody complexes’, which are rather large and tend to get stuck in the soft tissues and joints, causing inflammation and tissue damage [14]. If you get ‘arthritis’ after a vaccine or illness, now you know why! Antibodies!


True immunity requires a robust innate immune system (also known as Th1 immunity). This is the very first line of defence. As already mentioned, vaccines target antibody production, which is part of the humoral immune system (also known as Th2 immunity) – and the last function called into play by the immune system.

We can look upon these two arms of the immune system (innate and humoral) as being antagonistic – when one is dominant, the other is suppressed. So, a dominant antibody response (caused/exacerbated by repeat vaccinations), means that the innate immune system (first line of defence) is suppressed, leaving you more vulnerable to infection [15].

It should be noted here, that the disease known as ‘AIDS’ is characterised by this very same thing – high antibody counts, and poor function of the innate immune system [16]

Also of note – studies have shown that cancer and autism patients have this particular immune imbalance – high antibody counts and suppressed innate immunity [17-20].


Antibodies are extracellular, meaning that they are active outside the cells, but cannot actually enter cells…although scientists are trying to genetically engineer antibodies that will do just that [21].

Now, this is quite a conundrum, because antibodies are not called into action until after a pathogen has entered the cells, and antibodies can only bind to antigens on the surface of the cell (NOT inside the cell).

Now you have to rely on T-cells to orchestrate the killing of infected cells, in order to stop the spread of infection – this is the realm of the innate immune system (the one that is suppressed by repeated vaccinations, remember?). Such is the natural sequence of events when a th1-type response is generated, such as seen in natural infection [22].

The natural Th-1 type response is to eliminate infection via externalising it – this is the classic disease symptoms we know so well, such as rash, fever, cough, mucus, swelling etc [23]. Th2 dominance inhibits this natural response, which inevitably must lead to either:

  • altered disease manifestation, so for example, the vaccinated person who has whooping cough, may have a cough, but without the tell-tale ‘whoop’ sound [24].
  • chronic underlying infection, inflammation or auto-immune disease [25-26].

Let’s just re-emphasize that last point, because it’s really important, and once understood, you’ll never again look at vaccines the same way again…

First: Vaccines are designed to stimulate antibody production (Th2 immune system).

Second: Antibodies cannot stop infection, nor can they enter cells that are infected.

Third: Due to immune imbalance caused by vaccination, infected cells harbour infection chronically, causing inflammation and auto-immune conditions.

Fourth: person shows only mild or no signs of acute illness, but becomes progressively burdened down by chronic health issues.

So, what actually happens is that the vaccine has not prevented infection, it has simply prevented the body from expelling the infection.

It goes without saying, that such a state of affairs does wonders for the vaccine ‘efficacy’ statistics, since the vaccinated are less likely to show overt signs of acute disease, and therefore, less likely to be diagnosed, or even tested – meanwhile, chronic ‘non-communicable’ diseases continue to spiral out of control…

Now you know why.


[1] Crone NE, Reder AT. Severe tetanus in immunized patients with high anti-tetanus titers, Neurology, 1992, 42(4): 761-764.

[2] Maselle SY, Matre R, Mbise R, Hofstad T. Neonatal tetanus despite protective serum antitoxin concentration, FEMS Microbiol Immunol, 1991, 3(3): 171-175.

[3] Pitisuttithum P, Gilbert P, Gurwith M, et al. Randomized, double-blind, placebo-controlled efficacy trial of a bivalent recombinant glycoprotein 120 HIV-1 vaccine among injection drug users in Bangkok, Thailand. J Infect Dis, 2006, 194(12):1661-1761.

[4] Brodie M, Park W. Active Immunization Against Poliomyelitis, Am J Pub Health, 1936, 26:119–125.

[5] O’Connor A, Merrill W Chase, 98, Scientist Who Advanced Immunology, New York Times, Jan 22, 2004. Accessed October, 2018.

[6] Røsjø, E., Lossius, A., Abdelmagid, N., Lindstrøm, J. C., Kampman, M. T., Jørgensen, L., … Holmøy, T. (2017). Effect of high-dose vitamin D3 supplementation on antibody responses against Epstein–Barr virus in relapsing-remitting multiple sclerosis. Multiple Sclerosis Journal, 23(3), 395–402.

[7] Halstead SB, O’Rourke EJ. Antibody-enhanced dengue virus infection in primate leukocytes, Nature, 1977, 265(5596):739-741.

[8] ] Dejnirattisai W, Jumnainsong A, Onsirisakul N, et al. Cross-reacting antibodies enhance dengue virus infection in humans, Science, 2010, 328(5979):745-748.

[9] Dejnirattisai W, Supasa P, Wongwiwat W, et al. Dengue virus sero-cross-reactivity drives antibody-dependent  enhancement of infection with zika virus, Nat Immunol, 2016, 17(9):1102-1108.

[10] Homsy J, Meyer M, Tateno M, et al. The fc and not CD4 receptor mediates antibody enhancement of HIV infection in human cells, Science, 1989, 244(4910):1357+.

[11] Furuyama W, Marzi A, Carmody AB, et al. Fcy-receptor Ila-mediated Src signaling pathway is essential for the antibody-dependent enhancement of ebola virus infection, PLoS Pathogen, 2016, 12(12):e1006139.

[12] Biryukov S, Angov E, Landmesser ME, et al. Complement and antibody-mediated enhancement of red blood cell invasion and growth of malaria parasites, EBioMedicine, 2016, 9:207-216.

[13] Janeway CA Jr, Travers P, Walport M, et al. Immunobiology: The Immune System in Health and Disease. 5th edition. New York: Garland Science; 2001

[14] Cochrane CG, Dixon FJ. Cell and tissue damage through antigen-antibody complexes. Calif Med. 1969;111(2):99-112.

[15] Brad Spellberg, John E. Edwards; Type 1/Type 2 Immunity in Infectious Diseases, Clinical Infectious Diseases, Volume 32, Issue 1, 1 January 2001, Pages 76–102.

[16] Kaur R, Dhakad MS, Goyal R, Bhalla P, Dewan R (2016) Study of TH1/TH2 Cytokine Profiles in HIV/AIDS Patients in a Tertiary Care Hospital in India. J Med Microb Diagn 5:214

[17] Sato M, Goto S, Kaneko R, et al. Impaired production of Th1 cytokines and increased frequency of Th2 subsets in pBMC from advanced cancer patients. Anticancer Res, 1998, 18:3951-3955.

[18] Huang M, Wang J, Lee p, et al. Human non-small cell lung cancer cells express a type 2 cytokine pattern. Cancer Res, 1995, 55:3847-3853.

[19] Filella X, Alcover J, Zarco MA, et al. Analysis of type T1 and T2 cytokines in patients with prostate cancer, prostate, 2000, 44:271-274.

[20] Gupta, S., Aggarwal, S., Rashanravan, B., Lee, T., TH1 and TH2-like cytokines in CD4+ and CD8+ T cells in autism, J of Neuroimmunol, 1998; 85:106-109.

[21] Coghlan A. Super-antibodies break the cell barrier, New Scientist, Accessed December 2018.

[22] Kim EJ, Cho D, Kim TS. Efficient induction of T helper type 1-mediated immune responses in antigen-primed mice by anti-CD3 single-chain Fv/interleukin-18 fusion DNA, Immunology, 2004, 111(1): 27–34.

[23] Abbas AK, Murphy KM, Sher A. Functional diversity of helper T lymphocytes, Nature, 1996, 383(6603):787-93.

[24] Nelson KE, Williams C. Infectious Disease Epidemiology: Theory and practice 2007), Jones and Bartlett Learning, pp 131.

[25] Hayflick, L. Slow Viruses, Executive Health Report, Feb. 1981, pp 4.

[26] Talai, N., “Autoimmunity,” in Fudenberg, Basic Clinical Immunology, 3rd Ed., Lange, 1980, p. 222.